109 Zina Pitcher Place, Room 2065
Ann Arbor, MI 48109
Available to mentor
Thomas E. Wilson, M.D. Ph.D., is a Professor of Pathology and Human Genetics at the University of Michigan Medical School.
Dr. Wilson is a molecular genetic pathologist active in the clinic with related research interests that include basic studies of yeast double-strand break repair, mechanisms of mammalian chromosomal instability, genomic technologies in core facilities, and bioinformatics, including projects aimed at standardizing and sharing data analysis tools.
Education and mentorship of diverse trainees is critical to our mission and Dr. Wilson directs classes and UROP and other research projects related to molecular genetics, translational research, and bioinformatics.
Please visit https://wilsonte-umich.github.io/ learn more about Dr. Wilson's goals to advance understanding of the genetic processes that cause mutations associated with human diseases by detecting them in the clinical laboratory, exploring their mechanisms in the research laboratory, and developing resources to help students and other researchers do the same.
https://wilsonte-umich.github.io/
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ResidencyWashington University in St. Louis School of Medicine, Laboratory Medicine, 1999
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Postdoctoral FellowWashington University in St. Louis School of Medicine, St Louis, 1999
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Chief ResidentWashington University in St. Louis School of Medicine, St Louis, 1999
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Center MemberRogel Cancer Center
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Center MemberCenter for Computational Medicine and Bioinformatics
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Wilson TE, Ahmed S, Winningham A, Glover TW. Nat Commun, 2024 Nov 6; 15 (1): 9582Journal ArticleReplication stress induces POLQ-mediated structural variant formation throughout common fragile sites after entry into mitosis.
DOI:10.1038/s41467-024-53917-8 PMID: 39505880 -
Bedi K, Magnuson B, Narayanan IV, McShane A, Ashaka M, Paulsen MT, Wilson TE, Ljungman M. 2024 Jun 12;PreprintIsoform and pathway-specific regulation of post-transcriptional RNA processing in human cells.
DOI:10.1101/2024.06.12.598705 PMID: 38915566 -
Wilson TE, Ahmed S, Winningham A, Glover TW. bioRxiv,PreprintPOLQ mediates replication-stress induced structural variant formation throughout common fragile sites during mitosis
DOI:10.1101/2024.05.28.596214 -
McShane A, Narayanan IV, Paulsen MT, Ashaka M, Blinkiewicz H, Yang NT, Magnuson B, Bedi K, Wilson TE, Ljungman M. 2024 May 3;PreprintCharacterizing nascent transcription patterns of PROMPTs, eRNAs, and readthrough transcripts in the ENCODE4 deeply profiled cell lines.
DOI:10.1101/2024.04.09.588612 PMID: 38645116 -
Kim Y, Cheng W, Cho C-S, Hwang Y, Si Y, Park A, Schrank M, Hsu J-E, Anacleto A, Xi J, Kim M, Pedersen E, Koues OI, Wilson T, Lee C, Jun G, Kang HM, Lee JH. Nat Protoc, 2024 Oct 31;Journal ArticleSeq-Scope: repurposing Illumina sequencing flow cells for high-resolution spatial transcriptomics.
DOI:10.1038/s41596-024-01065-0 PMID: 39482362 -
Laufer VA, Glover TW, Wilson TE. Mutat Res Rev Mutat Res, 2023 792: 108475Journal ArticleApplications of advanced technologies for detecting genomic structural variation.
DOI:10.1016/j.mrrev.2023.108475 PMID: 37931775 -
Wilson TE, Ahmed S, Higgins J, Salk JJ, Glover TW. NAR Genom Bioinform, 2023 Jun; 5 (2): lqad042Journal ArticlesvCapture: efficient and specific detection of very low frequency structural variant junctions by error-minimized capture sequencing.
DOI:10.1093/nargab/lqad042 PMID: 37181851 -
Moran JV, Wilson TE. Cell, 2022 Sep 29; 185 (20): 3643 - 3645.Journal ArticleReverse transcriptase meets DNA, again: Possible roles for transposable elements in host DNA repair.
DOI:10.1016/j.cell.2022.09.012 PMID: 36179663