Toxoplasma gondii is a parasite that infects nearly all warm-blooded animals, with about one-third of the human population chronically infected. After infection, the active form, tachyzoites, disseminates rapidly throughout the body, causing tissue damage. While a healthy immune system can clear most tachyzoites, some evade detection and transform into chronic, latent cysts known as bradyzoites. These bradyzoite cysts are resistant to both immune clearance and treatment, posing an ongoing risk as they can reactivate during periods of immune suppression. Reactivation can lead to severe complications, including ocular lesions, brain lesions, and life-threatening diseases, particularly in immunocompromised individuals and developing fetuses. The precise molecular mechanisms governing bradyzoite formation remain incompletely understood. My research focuses on elucidating how the protein translation process regulates the differentiation of Toxoplasma gondii.