Available to mentor
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Associate ProfessorUniversity of the Ryukyus, School of Medicine, 2023
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Assistant ProfessorUniversity of Maryland, Baltimore, Anatomy and Neurobiology, 2020
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Director of Rodent behavior coreCase Western Reserve University, Cleveland, 2018
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Visiting lecturerConcordia College - Minnesota, Psychology, 2014
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Postdoctoral fellowUniversity of Maryland, Baltimore, Baltimore, 2013
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Research associateBinghamton University, Psychology, 2010
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Postdoctoral fellowUniversity of Hawaii at Manoa, Honolulu, 2007
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PhDNagoya University, Nagoya, 2006
I have recently moved from Japan as an Associate Professor of Physiology to U-M, as an Assistant Research Scientist in the Department of Pharmacology, to ensure my enthusiasm for research proceed further. I have a broad background in psychology and neuroscience, with specific training and expertise in behavioral mechanisms and circuit-manipulation in rodent models. My research is focused on neural circuits dynamics regulating sociability that sustains amicable social life and deficiency relevant to autism spectrum disorders (ASD). Using mainly mouse models, I have pursued to uncover 1) how prosocial (i.e., positive social) relationship can sustain development of neural circuits and mental health, 2) how ensemble of neural circuits determines sequence of natural social behaviors and 3) responsible decifiency for behavioral symptoms of ASD.
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Arakawa H, Tokashiki M. Eur J Neurosci, 2024 Oct; 60 (7): 5731 - 5749.Journal ArticleThe posterior intralaminar thalamic nucleus promotes nose-to-nose contacts leading to prosocial reception in the sequence of mouse social interaction.
DOI:10.1111/ejn.16520 PMID: 39210622 -
Arakawa H, Tokashiki M, Higuchi Y, Konno T. Peptides, 2024 May; 175: 171178Journal ArticleAdolescent social isolation disrupts developmental tuning of neuropeptide circuits in the hypothalamus to amygdala regulating social and defensive behavior.
DOI:10.1016/j.peptides.2024.171178 PMID: 38368908 -
Arakawa H. 2024 The Palgrave Encyclopedia of Disability, 1 - 7.ChapterAssessing Disability in Rodent Models of Human Disorders
DOI:10.1007/978-3-031-40858-8_104-1 -
Arakawa H, Higuchi Y. 2024 Jan 1; Neurobiology of Autism Spectrum Disorders, 105 - 121.ChapterThe Usability of Mouse Models to Study the Neural Circuity in Autism Spectrum Disorder: Regulatory Mechanisms of Core Behavioral Symptoms
DOI:10.1007/978-3-031-42383-3_6 -
Arakawa H. Physiol Behav, 2023 Dec 1; 272: 114373Journal ArticleRevisiting sociability: Factors facilitating approach and avoidance during the three-chamber test.
DOI:10.1016/j.physbeh.2023.114373 PMID: 37805136 -
Higuchi Y, Tada T, Nakachi T, Arakawa H. Neuropharmacology, 2023 Oct 1; 237: 109634Journal ArticleSerotonergic circuit dysregulation underlying autism-related phenotypes in BTBR mouse model of autism.
DOI:10.1016/j.neuropharm.2023.109634 PMID: 37301467 -
Higuchi Y, Tachigori S-I, Arakawa H. Psychoneuroendocrinology, 2023 Mar; 149: 106004Journal ArticleFaded neural projection from the posterior bed nucleus of the stria terminalis to the lateral habenula contributes to social signaling deficit in male BTBR mice as a mouse model of autism.
DOI:10.1016/j.psyneuen.2022.106004 PMID: 36543023 -
Arakawa H, Higuchi Y, Ozawa A. Res Sq, 2023 Mar 1;Journal ArticleOxytocin neurons in the paraventricular nucleus of the hypothalamus circuit-dependently regulates social behavior, which malfunctions in BTBR mouse model of autism.
DOI:10.21203/rs.3.rs-2621359/v1 PMID: 36909537
